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The vast majority of evidence comes from animal studies, which are promising but cannot be directly extrapolated to human outcomes

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The medicinal-chemistry programme that produced cagrilintide, described by Kruse and colleagues in the Journal of Medicinal Chemistry in 2021, addressed this by re-engineering the amylin backbone with structural elements borrowed from calcitonin (a related, non-aggregating peptide) and attaching a fatty-acid side chain that enables reversible binding to serum albumin [1]

Influence of PARP-1 inhibition in the cardiotoxicity of the topoisomerase 2 inhibitors doxorubicin and mitoxantrone

These changes were partially moderated by changes in brain morphometry, underscoring the interrelationship between weight and brain structure/ function